https://nova.newcastle.edu.au/vital/access/ /manager/Index ${session.getAttribute("locale")} 5 https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:16917 Tue 31 Jul 2018 16:15:02 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:5569 Sat 24 Mar 2018 07:49:28 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:42174 2⁻) production in senescent fibroblasts. Targeting STAT3 activity using the small-molecule inhibitor STA-21 attenuated IL-6 production, reduced p21 levels, decreased senescence-associated β-galactosidase accumulation, and restored normal mitochondrial function. The results of this study illustrate that stress-induced senescence in lung fibroblasts involves the activation of STAT3, which can be pharmacologically modulated.]]> Fri 26 Aug 2022 08:10:19 AEST ]]>